Two novel DnaJ chaperone proteins CG5001 and P58IPK regulate the pathogenicity of Huntington’s disease related aggregates-Reference-Cited by-同舟云学术

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Two novel DnaJ chaperone proteins CG5001 and P58IPK regulate the pathogenicity of Huntington’s disease related aggregates

Published:2024-09-06 Issue:1 Volume:14 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Deo Ankita,Ghosh Rishita,Ahire Snehal,Marathe Sayali,Majumdar Amitabha,Bose Tania

Funder

Indian Council of Medical Research

Department of Biotechnology, Ministry of Science and Technology, India

Wellcome Trust DBT India Alliance

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41598-024-71065-3.pdf

Reference95 articles.

1. Kovacs, G. Molecular pathological classification of neurodegenerative diseases: Turning towards precision medicine. Int. J. Mol. Sci. 17, 189. https://doi.org/10.3390/ijms17020189 (2016).

2. Brown, R. C., Lockwood, A. H. & Sonawane, B. R. Neurodegenerative diseases: An overview of environmental risk factors. Environ. Health Perspect. 113, 1250–1256. https://doi.org/10.1289/ehp.7567 (2005).

3. Sathasivam, S., Grierson, A. J. & Shaw, P. J. Characterization of the caspase cascade in a cell culture model of SOD1-related familial amyotrophic lateral sclerosis: Expression, activation and therapeutic effects of inhibition. Neuropathol. Appl. Neurobiol. 31, 467–485. https://doi.org/10.1111/j.1365-2990.2005.00658.x (2005).

4. Borthwick, G. M., Johnson, M. A., Ince, P. G., Shaw, P. J. & Turnbull, D. M. Mitochondrial enzyme activity in amyotrophic lateral sclerosis: implications for the role of mitochondria in neuronal cell death. Ann. Neurol. 46, 787–790. https://doi.org/10.1002/1531-8249(199911)46:5%3c787::aidana17%3e3.0.co;2-8 (1999).

5. Williams, R. E. et al. Cultured glial cells are resistant to the effects of motor neurone disease-associated SOD1 mutations. Neurosci. Lett. 302, 146–150. https://doi.org/10.1016/S0304-3940(01)01686-X (2001).

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