Evaluation of Keratinocyte Proliferation and Differentiation in Vitamin D Receptor Knockout Mice*
Author:
Affiliation:
1. Endocrine Unit, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02114
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/141/6/2043/10360962/endo2043.pdf
Reference28 articles.
1. The nuclear vitamin D receptor: biological and molecular regulatory properties revealed.;Haussler;J Bone Miner Res,1998
2. Normalization of mineral ion homeostasis by dietary means prevents hyperparathyroidism, rickets, and osteomalacia, but not alopecia in vitamin D receptor-ablated mice.;Li;Endocrinology,1998
3. Mice lacking the vitamin D receptor exhibit impaired bone formation, uterine hypoplasia and growth retardation after weaning.;Yoshizawa;Nat Genet,1997
4. Vitamin D, calcium, and epidermal differentiation.;Bikle;Endocr Rev,1993
5. The epidermal keratinocyte as a model for the study of gene regulation and cell differentiation.;Eckert;Physiol Rev,1997
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