Mice lacking the vitamin D receptor exhibit impaired bone formation, uterine hypoplasia and growth retardation after weaning

Author:

Yoshizawa Tatsuya,Handa Yuki,Uematsu Yoshikatsu,Takeda Shu,Sekine Keisuke,Yoshihara Yuko,Kawakami Toru,Arioka Kouki,Sato Hiroshi,Uchiyama Yasushi,Masushige Shoichi,Fukamizu Akiyoshi,Matsumoto Toshio,Kato Shigeaki

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Reference30 articles.

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2. Studzinski, G.P., McLane, J.A. & Uskokovic, M.R. Signaling pathways for vitamin D-induced differentiation: implications for therapy of proliferative and neoplastic diseases. CRC Crit. Rev. Eukaryotic Gene Expression 3, 279–312 (1993).

3. Walters, M.R. Newly identified actions of the vitamin D endocrine system. Endocr. Rev. 13, 719–764 (1992).

4. Darwish, H. & DeLuca, H.F., D–regulated gene expression. CRC Crit. Rev. Eukaryotic Gene Expression 3, 89–116 (1993).

5. Kato, S. et al. Widely spaced, directly repeated PuGGTCA elements act as promiscuous enhancers for different classes of nuclear receptors. Mol. Cell. Biol. 15, 5858–5867 (1995).

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