A subset of <scp>OPCs</scp> do not express Olig2 during development which can be increased in the adult by brain injuries and complex motor learning-Reference-Cited by-同舟云学术

A subset of OPCs do not express Olig2 during development which can be increased in the adult by brain injuries and complex motor learning

Published:2022-10-29 Issue:2 Volume:71 Page:415-430
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Fang Li‐Pao¹^ORCID,Liu Qing¹^ORCID,Meyer Erika¹²^ORCID,Welle Anna³^ORCID,Huang Wenhui¹^ORCID,Scheller Anja¹^ORCID,Kirchhoff Frank¹⁴^ORCID,Bai Xianshu¹^ORCID

Affiliation:

1. Molecular Physiology, Center for Integrative Physiology and Molecular Medicine University of Saarland Homburg Germany

2. Laboratory of Brain Ischemia and Neuroprotection, Department of Pharmacology and Therapeutics State University of Maringá Maringá Brazil

3. Department of Genetics and EpiGenetics University of Saarland Saarbrücken Germany

4. Experimental Research Center for Normal and Pathological Aging University of Medicine and Pharmacy of Craiova Craiova

Funder

Deutsche Forschungsgemeinschaft

Unitatea Executiva pentru Finantarea Invatamantului Superior, a Cercetarii, Dezvoltarii si Inovarii

Universität des Saarlandes

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.24284

Reference64 articles.

1. After traumatic brain injury oligodendrocytes regain a plastic phenotype and can become astrocytes

2. Astrocyte uncoupling as a cause of human temporal lobe epilepsy

3. Glutamatergic synapses on oligodendrocyte precursor cells in the hippocampus

4. Expression pattern of the transcription factor Olig2 in response to brain injuries: Implications for neuronal repair

5. Hippocampal connectivity with sensorimotor cortex during volitional finger movements: Laterality and relationship to motor learning

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