ADP‐heptose enables Helicobacter pylori to exploit macrophages as a survival niche by suppressing antigen‐presenting HLA‐II expression
Author:
Affiliation:
1. Department of Biology University of Padova Italy
2. Department of Experimental and Clinical Medicine University of Florence Italy
3. Medical Microbiology and Hospital Epidemiology Max von Pettenkofer Institute LMU Munich Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/1873-3468.14156
Reference26 articles.
1. Immune evasion strategies used byHelicobacter pylori
2. Helicobacter pylori Dampens HLA-II Expression on Macrophages via the Up-Regulation of miRNAs Targeting CIITA
3. Helicobacter pylori phagosome maturation in primary human macrophages
4. Helicobacter pylori Affects the Antigen Presentation Activity of Macrophages Modulating the Expression of the Immune Receptor CD300E through miR-4270
5. Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island
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4. Innate activation of human neutrophils and neutrophil-like cells by the pro-inflammatory bacterial metabolite ADP-heptose and Helicobacter pylori;International Journal of Medical Microbiology;2023-07
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