Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island

Author:

Viala Jérôme,Chaput Catherine,Boneca Ivo G,Cardona Ana,Girardin Stephen E,Moran Anthony P,Athman Rafika,Mémet Sylvie,Huerre Michel R,Coyle Anthony J,DiStefano Peter S,Sansonetti Philippe J,Labigne Agnès,Bertin John,Philpott Dana J,Ferrero Richard L

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Reference50 articles.

1. Parsonnet, J. Helicobacter pylori: the size of the problem. Gut 43, S6–S9 (1998).

2. Censini, S. et al. cag, a pathogenicity island of Helicobacter pylori, encodes type Ispecific and disease-associated virulence factors. Proc. Natl Acad. Sci. USA 93, 14648–14653 (1996).

3. Blaser, M.J. et al. Infection with Helicobacter pylori strains possessing cagA is associated with an increased risk of developing adenocarcinoma of the stomach. Cancer Res. 55, 2111–2115 (1995).

4. Peek, R.M. Jr . et al. Heightened inflammatory response and cytokine expression in vivo to cagA+Helicobacter pylori strains. Lab. Invest. 73, 760–770 (1995).

5. Fischer, W. et al. Systematic mutagenesis of the Helicobacter pylori cag pathogenicity island: essential genes for CagA translocation in host cells and induction of interleukin-8. Mol. Microbiol. 42, 1337–1348 (2001).

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