The evolution of arsenic in the treatment of acute promyelocytic leukemia and other myeloid neoplasms: Moving toward an effective oral, outpatient therapy
Author:
Affiliation:
1. Department of Leukemia; The University of Texas MD Anderson Cancer Center; Houston Texas
Funder
National Cancer Institute
Publisher
Wiley
Subject
Cancer Research,Oncology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/cncr.29852/fullpdf
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3. In vitro studies on cellular and molecular mechanisms of arsenic trioxide (As203) in the treatment of acute promyelocytic leukemia: As203 induces NB4 cell apoptosis with downregulation of Bcl-2 expression and modulation of PML-RAR alpha/PML proteins;Chen;Blood,1996
4. The t(15;17) translocation in acute promyelocytic laeukemia fuses the retinoic acid receptor α gene to another transcribed locus;The;Nature,1990
5. PML protein expression in hematopoietic and acute promyelocytic leukemia cells;Daniel;Blood,1993
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