Costunolide promotes imatinib-induced apoptosis in chronic myeloid leukemia cells via the Bcr/Abl-Stat5 pathway
Author:
Affiliation:
1. Clinical Laboratory; The Second Hospital of Dalian Medical University; Dalian 116023 China
Funder
The Basic Research Program of Higher Education of Liaoning Province of China
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/ptr.6106/fullpdf
Reference22 articles.
1. Enhanced targeting of CML stem and progenitor cells by inhibition of porcupine acyltransferase in combination with TKI;Agarwal;Blood,2017
2. Bcr-Abl expression levels determine the rate of development of resistance to imatinib mesylate in chronic myeloid leukemia;Barnes;Cancer Research,2005
3. PAK-dependent STAT5 serine phosphorylation is required for BCR-ABL-induced leukemogenesis;Berger;Leukemia,2014
4. Two naturally occurring terpenes, dehydrocostuslactone and costunolide, decrease intracellular GSH content and inhibit STAT3 activation;Butturini;PLoS One,2011
5. Dehydrocostus lactone suppresses proliferation of human chronic myeloid leukemia cells through Bcr/Abl-JAK/STAT signaling pathways;Cai;Journal of Cellular Biochemistry,2017
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