<scp> <i>UBTF</i> ‐internal </scp> tandem duplication as a novel poor prognostic factor in pediatric acute myeloid leukemia-Reference-Cited by-同舟云学术

UBTF ‐internal tandem duplication as a novel poor prognostic factor in pediatric acute myeloid leukemia

Published:2022-12-07 Issue:4 Volume:62 Page:202-209
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosomes & Cancer

Author:

Kaburagi Taeko¹²,Shiba Norio³,Yamato Genki¹²,Yoshida Kenichi⁴,Tabuchi Ken⁵,Ohki Kentaro⁶^ORCID,Ishikita Etsuko¹²,Hara Yusuke²^ORCID,Shiraishi Yuichi⁷,Kawasaki Hirohide¹,Sotomatsu Manabu¹,Takizawa Takumi²,Taki Tomohiko⁸,Kiyokawa Nobutaka⁶^ORCID,Tomizawa Daisuke⁹^ORCID,Horibe Keizo¹⁰,Miyano Satoru¹¹,Taga Takashi¹²,Adachi Souichi¹³,Ogawa Seishi⁴¹⁴¹⁵,Hayashi Yasuhide¹¹⁶

Affiliation:

1. Department of Hematology/Oncology Gunma Children's Medical Center Gunma Japan

2. Department of Pediatrics Gunma University Graduate School of Medicine Gunma Japan

3. Department of Pediatrics Yokohama City University Graduate School of Medicine Kanagawa Japan

4. Department of Pathology and Tumor Biology, Graduate School of Medicine Kyoto University Kyoto Japan

5. Department of Pediatrics Tokyo Metropolitan Cancer and Infectious Diseases Center Komagome Hospital Tokyo Japan

6. Department of Pediatric Hematology and Oncology Research National Research institute for Child Health and Development Tokyo Japan

7. Division of Genome Analysis Platform Development National Cancer Center Research Institute Tokyo Japan

8. Department of Medical Technology Kyorin University Faculty of Health Sciences Tokyo Japan

9. Division of Leukemia and Lymphoma Children's Cancer Center, National Center for Child Health and Development Tokyo Japan

10. Clinical Research Center National Hospital Organization Nagoya Medical Center Aichi Japan

11. M&D Data Science Center Tokyo Medical and Dental University Tokyo Japan

12. Department of Pediatrics Shiga University of Medical Science Shiga Japan

13. Human Health Science, Graduate School of Medicine Kyoto University Kyoto Japan

14. Department of Medicine Center for Hematology and Regenerative Medicine, Karolinska Institute Stockholm Sweden

15. Institute for the Advanced Study of Human Biology (WPI‐ASHBi) Kyoto University Kyoto Japan

16. Institute of Physiology and Medicine Jobu University Gunma Japan

Publisher

Wiley

Subject

Cancer Research,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/gcc.23110

Reference15 articles.

1. The molecular landscape of pediatric acute myeloid leukemia reveals recurrent structural alterations and age-specific mutational interactions

2. Tandem duplications ofMLLandFLT3are correlated with poor prognoses in pediatric acute myeloid leukemia: A study of the Japanese childhood AML Cooperative Study Group

3. FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

4. Favorable outcome of patients with acute myeloid leukemia harboring a low-allelic burden FLT3-ITD mutation and concomitant NPM1 mutation: relevance to post-remission therapy

5. Transcriptome analysis offers a comprehensive illustration of the genetic background of pediatric acute myeloid leukemia

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1. UBTFTandem Duplications in Pediatric MDS and AML: Implications for Clinical Screening and Diagnosis;2023-11-13

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3. UBTF tandem duplications are rare but recurrent alterations in adult AML and associated with younger age, myelodysplasia, and inferior outcome;Blood Cancer Journal;2023-05-26

4. UBTF tandem duplications define a distinct subtype of adult de novo acute myeloid leukemia;Leukemia;2023-04-21