Recurrent mutation ofJAK3in T-cell prolymphocytic leukemia-Reference-Cited by-同舟云学术

Recurrent mutation ofJAK3in T-cell prolymphocytic leukemia

Published:2014-01-21 Issue:4 Volume:53 Page:309-316
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosomes Cancer

Author:

Bergmann Anke K.¹²,Schneppenheim Sina¹,Seifert Marc³,Betts Matthew J.⁴,Haake Andrea¹,Lopez Cristina¹,Maria Murga Penas Eva¹,Vater Inga¹,Jayne Sandrine⁵,Dyer Martin J.S.⁵,Schrappe Martin²,Dührsen Ulrich⁶,Ammerpohl Ole¹,Russell Robert B.⁴,Küppers Ralf³,Dürig Jan⁶,Siebert Reiner¹

Affiliation:

1. Institute for Human Genetics; Christian-Albrechts-University Kiel and University Hospital Schleswig-Holstein; Campus Kiel Kiel Germany

2. Department of Pediatrics; Christian-Albrechts-University Kiel and University Hospital Schleswig-Holstein; Campus Kiel Kiel Germany

3. Institute of Cell Biology (Cancer Research); Medical School Essen, University of Duisburg-Essen; Essen Germany

4. Cell Networks Excellenz Cluster; Bioquant, University of Heidelberg; Heidelberg Germany

5. The MRC Toxicology Unit; University of Leicester; Leicester UK

6. Department of Hematology; University Hospital Essen, University of Duisburg-Essen; Essen Germany

Publisher

Wiley

Subject

Cancer Research,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/gcc.22141/fullpdf

Reference44 articles.

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2. T cell development and activation in Jak3-deficient mice;Baird;J Leukoc Biol,1998

3. Crystal structures of the JAK2 pseudokinase domain and the pathogenic mutant V617F;Bandaranayake;Nat Struct Mol Biol,2012

4. Recurrent JAK1 and JAK3 somatic mutations in T-cell prolymphocytic leukemia;Bellanger;Leukemia.,2013

5. JAK3 deregulation by activating mutations confers invasive growth advantage in extranodal nasal-type natural killer cell lymphoma;Bouchekioua;Leukemia,2013

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