Defective PITRM 1 mitochondrial peptidase is associated with Aβ amyloidotic neurodegeneration-Reference-Cited by-同舟云学术

Defective PITRM 1 mitochondrial peptidase is associated with Aβ amyloidotic neurodegeneration

Published:2015-12-23 Issue:3 Volume:8 Page:176-190
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Brunetti Dario¹,Torsvik Janniche²,Dallabona Cristina³,Teixeira Pedro⁴,Sztromwasser Pawel⁵⁶,Fernandez‐Vizarra Erika¹,Cerutti Raffaele¹,Reyes Aurelio¹,Preziuso Carmela⁷,D'Amati Giulia⁷,Baruffini Enrico³,Goffrini Paola³,Viscomi Carlo¹,Ferrero Ileana³,Boman Helge⁸,Telstad Wenche⁹,Johansson Stefan⁵⁸,Glaser Elzbieta⁴,Knappskog Per M⁵⁸,Zeviani Massimo¹,Bindoff Laurence A²¹⁰

Affiliation:

1. MRC Mitochondrial Biology Unit Wellcome Trust Cambridge UK

2. Department of Neurology Haukeland University Hospital Bergen Norway

3. Department of Life Sciences University of Parma Parma Italy

4. Department of Biochemistry and Biophysics Stockholm University Stockholm Sweden

5. Department of Clinical Science University of Bergen Bergen Norway

6. Computational Biology Unit Department of Informatics University of Bergen Bergen Norway

7. Department of Radiological Oncological and Pathological Sciences Sapienza University of Rome Rome Italy

8. Center for Medical Genetics and Molecular Medicine Haukeland University Hospital Bergen Norway

9. Department of Neurology Førde Hospital Førde Norway

10. Department of Clinical Medicine (K1) University of Bergen Bergen Norway

Funder

European Research Council

Vetenskapsrådet

Helse Vest

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201505894

Reference34 articles.

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2. Decreased Proteolytic Activity of the Mitochondrial Amyloid-β Degrading Enzyme, PreP Peptidasome, in Alzheimer's Disease Brain Mitochondria

3. Cerebrospinal Fluid β-Amyloid(1-42) in Alzheimer Disease

4. Pantethine treatment is effective in recovering the disease phenotype induced by ketogenic diet in a pantothenate kinase-associated neurodegeneration mouse model

5. The specificity of the staining of amyloid deposits with thioflavine T

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