Nuclear IL‐33/SMAD signaling axis promotes cancer development in chronic inflammation-Reference-Cited by-同舟云学术

Nuclear IL‐33/SMAD signaling axis promotes cancer development in chronic inflammation

Published:2021-02-22 Issue:7 Volume:40 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Park Jong Ho¹,Ameri Amir H¹,Dempsey Kaitlin E¹^ORCID,Conrad Danielle N¹,Kem Marina²,Mino‐Kenudson Mari²,Demehri Shadmehr¹^ORCID

Affiliation:

1. Center for Cancer Immunology and Cutaneous Biology Research Center Department of Dermatology Center for Cancer Research Massachusetts General Hospital and Harvard Medical School Boston MA USA

2. Department of Pathology Massachusetts General Hospital and Harvard Medical School Boston MA USA

Funder

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Burroughs Wellcome Fund

Sidney Kimmel Foundation

National Research Foundation of Korea

Howard Hughes Medical Institute

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2020106151

Reference49 articles.

1. The Dual Function Cytokine IL-33 Interacts with the Transcription Factor NF-κB To Dampen NF-κB–Stimulated Gene Transcription

2. IL-33/regulatory T cell axis triggers the development of a tumor-promoting immune environment in chronic inflammation

3. Pancreatitis is a risk factor for pancreatic cancer

4. Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer

5. Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer

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