Generation of a constitutive Na+-dependent inward-rectifier current in rat adult atrial myocytes by overexpression of Kir3.4
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2007.140772/fullpdf
Reference51 articles.
1. Acute desensitization of GIRK current in rat atrial myocytes is related to K+ current flow.;Bender;J Physiol,2004
2. Overexpression of monomeric and multimeric GIRK4 subunits in rat atrial myocytes removes fast desentitization and reduces inward rectification of muscarinic K+ current (IK(ACh)).;Bender;J Biol Chem,2001
3. Transfection of a phosphatidyl-4-phosphate 5-kinase gene into rat atrial myocytes removes inhibition of GIRK current by endothelin and α-adrenergic agonists.;Bender;FEBS Lett,2002
4. Contribution of the Kir3.1 subunit to the muscarinic-gated atrial potassium channel IKACh;Bettahi;J Biol Chem,2002
5. Alterations in potassium channel gene expression in atria of patients with persistent and paroxysmal atrial fibrillation: differential regulation of protein and mRNA levels of K+ channels;Brundel;J Am Coll Cardiol,2001
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