Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K + Channel) Channel Function-Reference-Cited by-同舟云学术

Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K + Channel) Channel Function

Published:2019-01 Issue:1 Volume:12 Page:
ISSN:2574-8300
Container-title:Circulation: Genomic and Precision Medicine
language:en
Short-container-title:Circ: Genomic and Precision Medicine

Author:

Kuß Johanna¹,Stallmeyer Birgit¹,Goldstein Matthias²,Rinné Susanne²,Pees Christiane³,Zumhagen Sven¹,Seebohm Guiscard¹,Decher Niels²,Pott Lutz⁴,Kienitz Marie-Cécile⁴,Schulze-Bahr Eric¹

Affiliation:

1. Department of Cardiovascular Medicine, Institute for Genetics of Heart Diseases (IfGH), University Hospital Münster, Germany (J.K., B.S., S.Z., G.S., E.S.-B.).

2. Institute of Physiology and Pathophysiology, Vegetative Physiology, University of Marburg, Germany (M.G., S.R., N.D.).

3. Department of Pediatric Cardiology, University Children’s Hospital Vienna, Austria (C.P.).

4. Department of Cardiovascular Medicine, Institute of Physiology, Ruhr-University Bochum, Germany (L.P., M.-C.K.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

General Medicine

Reference33 articles.

1. The Evaluation and Management of Bradycardia

2. Pacemaker channel dysfunction in a patient with sinus node disease

3. Familial Sinus Bradycardia Associated with a Mutation in the Cardiac Pacemaker Channel

4. Congenital sick sinus syndrome caused by recessive mutations in the cardiac sodium channel gene (SCN5A)

5. Catecholaminergic polymorphic ventricular tachycardia: RYR2 mutations, bradycardia, and follow up of the patients

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