Acute desensitization of GIRK current in rat atrial myocytes is related to K+current flow
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2004.072462/fullpdf
Reference50 articles.
1. Overexpression of monomeric and multimeric GIRK4 subunits in rat atrial myocytes removes fast desensitization and reduces inward rectification of muscarinic K+ current (IK(ACh));Bender;J Biol Chem,2001
2. Activation of muscarinic K+ current by β-adrenergic receptors in cultured atrial myocytes transfected with β1 subunit of heterotrimeric G proteins;Bender;FEBS Lett,1998
3. Transfection of a phosphatidyl-4-phosphate 5-kinase gene into rat atrial myocytes removes inhibition of GIRK current by endothelin and α-adrenergic agonists;Bender;FEBS Lett,2002
4. Transfection with 5-HT1A receptor gene and antisense directed against muscarinic M2 receptors reveal a mutual influence between Gi/o-coupled receptors in rat arial myoctes;Bösche;J Mol Cell Cardiol,2003a
5. G Protein-independent inhibition of GIRK current by adenosine in rat atrial myocytes overexpressing A1-adenosine receptors after adenovirus-mediated gene transfer;Bösche;J Physiol,2003b
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