A mutation protective against Alzheimer's disease renders amyloid β precursor protein incapable of mediating neurotoxicity-Reference-Cited by-同舟云学术

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A mutation protective against Alzheimer's disease renders amyloid β precursor protein incapable of mediating neurotoxicity

Published:2014-04-10 Issue:2 Volume:130 Page:291-300
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Hashimoto Yuichi¹,Matsuoka Masaaki¹

Affiliation:

1. Department of Pharmacology; Tokyo Medical University; Tokyo Japan

Funder

Grant-in-Aid for Scientific Research

Ministry of Education, Culture, Sports, Science, and Technology

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12717/fullpdf

Reference43 articles.

1. Amyloid precursor protein mediates a tyrosine kinase-dependent activation response in endothelial cells;Austin;J. Neurosci.,2009

2. The mechanism of γ-secretase dysfunction in familial Alzheimer disease;Chávez-Gutiérrez;EMBO J.,2012

3. Cdk5 deregulation in the pathogenesis of Alzheimer's disease;Cruz;Trends Mol. Med.,2004

4. A recessive mutation in the APP gene with dominant-negative effect on amyloidogenesis;Fede;Science,2009

5. Altered expression of transforming growth factor-β in Alzheimer's disease;Flanders;Neurology,1995

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2. The Protective A673T Mutation of Amyloid Precursor Protein (APP) in Alzheimer’s Disease;Molecular Neurobiology;2021-04-29

3. Hydration Thermodynamics of the N-Terminal FAD Mutants of Amyloid-β;Journal of Chemical Information and Modeling;2021-01-14

4. The PERK-Dependent Molecular Mechanisms as a Novel Therapeutic Target for Neurodegenerative Diseases;International Journal of Molecular Sciences;2020-03-19

5. Real-time nanoscale organization of amyloid precursor protein;Nanoscale;2020

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