Pridopidine reduces mutant huntingtin‐induced endoplasmic reticulum stress by modulation of the Sigma‐1 receptor-Reference-Cited by-同舟云学术

Pridopidine reduces mutant huntingtin‐induced endoplasmic reticulum stress by modulation of the Sigma‐1 receptor

Published:2021-04-28 Issue:2 Volume:158 Page:467-481
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Shenkman Marina¹²,Geva Michal³,Gershoni‐Emek Noga³,Hayden Michael R.³,Lederkremer Gerardo Z.¹²^ORCID

Affiliation:

1. The Shmunis School of Biomedicine and Cancer Research Cell Biology Division George Wise Faculty of Life Sciences Tel Aviv University Tel Aviv Israel

2. Sagol School of Neuroscience Tel Aviv University Tel Aviv Israel

3. Prilenia Therapeutics Development LTD Herzliya Israel

Funder

Israel Science Foundation

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.15366

Reference73 articles.

1. A mutation in sigma-1 receptor causes juvenile amyotrophic lateral sclerosis

2. Endoplasmic Reticulum (ER) Mannosidase I Is Compartmentalized and Required for N-Glycan Trimming to Man5–6GlcNAc2 in Glycoprotein ER-associated Degradation

3. Glycan regulation of ER-associated degradation through compartmentalization

4. Dysfunction in endoplasmic reticulum-mitochondria crosstalk underlies SIGMAR1 loss of function mediated motor neuron degeneration

5. Neuroprotective Effect of σ1-Receptors on the Cell Model of Huntington’s Disease

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