Interferon‐α promotes MHC I antigen presentation of islet β cells through STAT1‐IRF7 pathway in type 1 diabetes
Author:
Affiliation:
1. Department of Endocrinology and Metabolism The First Affiliated Hospital of Nanjing Medical University Nanjing China
2. Department of Pediatrics The First Affiliated Hospital of Nanjing Medical University Nanjing China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/imm.13468
Reference42 articles.
1. The impact of proinflammatory cytokines on the β-cell regulatory landscape provides insights into the genetics of type 1 diabetes
2. Viral infections in type 1 diabetes mellitus — why the β cells?
3. The Diabetes Susceptibility Gene Clec16a Regulates Mitophagy
4. Identification of non-HLA genes associated with development of islet autoimmunity and type 1 diabetes in the prospective TEDDY cohort
5. IFN-α Mediates the Development of Autoimmunity both by Direct Tissue Toxicity and through Immune Cell Recruitment Mechanisms
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1. Phosphorylation of aryl hydrocarbon receptor interacting protein by TBK1 negatively regulates IRF7 and the type I interferon response;Journal of Biological Chemistry;2024-01
2. Teplizumab: promises and challenges of a recently approved monoclonal antibody for the prevention of type 1 diabetes or preservation of residual beta cell function;Expert Review of Clinical Immunology;2023-11-12
3. The role of the interferon/JAK-STAT axis in driving islet HLA-I hyperexpression in type 1 diabetes;Frontiers in Endocrinology;2023-10-06
4. IRF7: role and regulation in immunity and autoimmunity;Frontiers in Immunology;2023-08-10
5. Single-cell RNA sequencing combined with single-cell proteomics identifies the metabolic adaptation of islet cell subpopulations to high-fat diet in mice;Diabetologia;2022-12-20
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