Are both protein kinase A‐ and protein kinase C‐dependent pathways involved in glucagon‐like peptide‐1 action on pancreatic insulin secretion?
Author:
Affiliation:
1. Oxford Center for Diabetes, Endocrinology and Metabolism Churchill Hospital Site Headington Oxford UK
2. Department of Diabetes, Endocrinology and Metabolism Kawasaki Medical School Kurashiki Okayama Japan
Publisher
Wiley
Subject
General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jdi.12225
Reference8 articles.
1. Low, but Physiological, Concentration of GLP-1 Stimulates Insulin Secretion Independent of the cAMP-Dependent Protein Kinase Pathway
2. Expression and functional activity of glucagon, glucagon‐like peptide I, and glucose‐dependent insulinotropic peptide receptors in rat pancreatic islet cells;Moens K;Diabetes,1996
3. Comparative effects of GLP-1 and GIP on cAMP production, insulin secretion, and in vivo antidiabetic actions following substitution of Ala8/Ala2 with 2-aminobutyric acid
4. Glucagon-Like Peptide-I Analogs: Effects on Insulin Secretion and Adenosine 3′,5′-Monophosphate Formation*
5. Physiological concentrations of GLP‐1 increase insulin secretion by activating protein kinase C pathway in pancreatic beta cells;Shigeto M;Diabetologia,2012
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