Hyperammonemia alters glycinergic neurotransmission and modulation of the glutamate-nitric oxide-cGMP pathway by extracellular glycine in cerebellum in vivo-Reference-Cited by-同舟云学术

Hyperammonemia alters glycinergic neurotransmission and modulation of the glutamate-nitric oxide-cGMP pathway by extracellular glycine in cerebellum in vivo

Published:2016-03-02 Issue:4 Volume:137 Page:539-548
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Cabrera-Pastor Andrea¹,Taoro-Gonzalez Lucas¹,Felipo Vicente¹

Affiliation:

1. Laboratorio de Neurobiología; Centro Investigación Príncipe Felipe de Valencia; Valencia Spain

Funder

Ministerio de Ciencia e Innovación

Consellería Educación Generalitat Valenciana

European Regional Development Funds

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13579/fullpdf

Reference45 articles.

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2. Metabotropic glutamate receptor 5 modulates the nitric oxide-cGMP pathway in cerebellum in vivo through activation of AMPA receptors;Boix;Neurochem. Int.,2011

3. Behavioral sensitization to cocaine is associated with increased AMPA receptor surface expression in the nucleus accumbens;Boudreau;J. Neurosci.,2005

4. Differential effects of chronic hyperammonemia on modulation of the glutamate-nitric oxide-cGMP pathway by metabotropic glutamate receptor 5 and low and high affinity AMPA receptors in cerebellum in vivo;Cabrera-Pastor;Neurochem. Int.,2012

5. Roles of the NMDA receptor and EAAC1 transporter in the modulation of extracellular glutamate by low and high affinity AMPA receptors in the cerebellum in vivo: differential alteration in chronic hyperammonemia;Cabrera-Pastor;ACS Chem. Neurosci.,2015

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