The S1PR2‐CCL2‐BDNF‐TrkB pathway mediates neuroinflammation and motor incoordination in hyperammonaemia-Reference-Cited by-同舟云学术

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The S1PR2‐CCL2‐BDNF‐TrkB pathway mediates neuroinflammation and motor incoordination in hyperammonaemia

Published:2022-02-24 Issue:4 Volume:48 Page:
ISSN:0305-1846
Container-title:Neuropathology and Applied Neurobiology
language:en
Short-container-title:Neuropathology Appl Neurobio

Author:

Arenas Yaiza M.¹,Balzano Tiziano¹,Ivaylova Gergana¹,Llansola Marta¹^ORCID,Felipo Vicente¹^ORCID

Affiliation:

1. Laboratory of Neurobiology Centro Investigación Príncipe Felipe Valencia Spain

Funder

Generalitat Valenciana

Ministerio de Ciencia e Innovación

Publisher

Wiley

Subject

Physiology (medical),Neurology (clinical),Neurology,Histology,Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/nan.12799

Reference89 articles.

1. Hepatic encephalopathy: effects of liver failure on brain function

2. Systemic inflammatory response exacerbates the neuropsychological effects of induced hyperammonemia in cirrhosis

3. Non invasive blood flow measurement in cerebellum detects minimal hepatic encephalopathy earlier than psychometric tests

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4. Increased levels and activation of the IL-17 receptor in microglia contribute to enhanced neuroinflammation in cerebellum of hyperammonemic rats;Biological Research;2024-04-27

5. Neuroinflammation alters GABAergic neurotransmission in hyperammonemia and hepatic encephalopathy, leading to motor incoordination. Mechanisms and therapeutic implications;Frontiers in Pharmacology;2024-03-15

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