Doubled lifespan and patient‐like pathologies in progeria mice fed high‐fat diet-Reference-Cited by-同舟云学术

Doubled lifespan and patient‐like pathologies in progeria mice fed high‐fat diet

Published:2018-12-12 Issue:1 Volume:18 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Kreienkamp Ray¹,Billon Cyrielle²,Bedia‐Diaz Gonzalo¹,Albert Carolyn J.¹,Toth Zacharie³,Butler Andrew A.⁴,McBride‐Gagyi Sara³,Ford David A.¹,Baldan Angel¹,Burris Thomas P.²,Gonzalo Susana¹

Affiliation:

1. Edward A. Doisy Department of Biochemistry and Molecular Biology St Louis University School of Medicine St Louis Missouri

2. Center for Clinical Pharmacology Washington University School of Medicine and St. Louis College of Pharmacy St Louis Missouri

3. Department of Orthopedic Surgery St Louis University School of Medicine St Louis Missouri

4. Department of Pharmacology and Physiology St Louis University School of Medicine St Louis Missouri

Funder

Congressionally Directed Medical Research Programs

National Institute on Aging

National Institute of General Medical Sciences

American Heart Association

National Institutes of Health

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12852

Reference36 articles.

1. Cancer cachexia: understanding the molecular basis

2. Lifespan extension by dietary intervention in a mouse model of Cockayne Syndrome uncouples early postnatal development from segmental progeria

3. IKKβ/NF-κB Activation Causes Severe Muscle Wasting in Mice

4. Rapamycin Reverses Cellular Phenotypes and Enhances Mutant Protein Clearance in Hutchinson-Gilford Progeria Syndrome Cells

5. A farnesyltransferase inhibitor prevents both the onset and late progression of cardiovascular disease in a progeria mouse model

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