Lifespan extension by dietary intervention in a mouse model of Cockayne Syndrome uncouples early postnatal development from segmental progeria
Author:
Affiliation:
1. Department of Genetics and Complex Diseases; Harvard School of Public Health; 655 Huntington Ave Boston MA 02115 USA
2. Department of Developmental Biology; Harvard School of Dental Medicine; 188 Longwood Avenue Boston MA 02115 USA
Publisher
Wiley
Subject
Cell Biology,Ageing
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12142/fullpdf
Reference22 articles.
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2. Premature aging in mice deficient in DNA repair and transcription;Boer;Science,2002
3. Cockayne's syndrome: literature review and case report;Boraz;Pediatr. Dent.,1991
4. Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic disease;Brooks;DNA Repair (Amst).,2013
5. Disorders of nucleotide excision repair: the genetic and molecular basis of heterogeneity;Cleaver;Nat. Rev. Genet.,2009
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