Luteinizing Hormone, a Reproductive Regulator That Modulates the Processing of Amyloid-β Precursor Protein and Amyloid-β Deposition-Reference-Cited by-同舟云学术

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Luteinizing Hormone, a Reproductive Regulator That Modulates the Processing of Amyloid-β Precursor Protein and Amyloid-β Deposition

Published:2004-05 Issue:19 Volume:279 Page:20539-20545
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Bowen Richard L.,Verdile Giuseppe,Liu Tianbing,Parlow Albert F.,Perry George,Smith Mark A.,Martins Ralph N.,Atwood Craig S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference77 articles.

1. Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein Precursor

2. Mutations associated with a locus for familial Alzheimer's disease result in alternative processing of amyloid beta-protein precursor

3. An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp 717 ) Mutants

4. High levels of amyloid-β protein from S182 (Glu246) familial Alzheimerʼs cells

5. Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

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