An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp 717 ) Mutants-Reference-Cited by-同舟云学术

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An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp 717 ) Mutants

Published:1994-05-27 Issue:5163 Volume:264 Page:1336-1340
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Suzuki Nobuhiro¹,Cheung Tobun T.²,Cai Xiao-Dan²,Odaka Asano¹,Otvos Laszlo³,Eckman Christopher²,Golde Todd E.²,Younkin Steven G.²

Affiliation:

1. Discovery Research Division, Takeda Chemical Industries, Ltd., Ibaraki, Japan.

2. Division of Neuropathology, Institute of Pathology, Case Western Reserve University, Cleveland, OH 44106, USA.

3. Wistar Institute of Anatomy and Biology, Philadelphia, PA 19104, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference49 articles.

1. ANWAR, N, LANCET 342: 1308 (1993).

2. BORGAONKAR, D.S., LINKAGE OF LATE-ONSET ALZHEIMERS-DISEASE WITH APOLIPOPROTEIN-E TYPE-4 ON CHROMOSOME-19, LANCET 342: 625 (1993).

3. BURDICK, D, ASSEMBLY AND AGGREGATION PROPERTIES OF SYNTHETIC ALZHEIMERS A4/BETA AMYLOID PEPTIDE ANALOGS, JOURNAL OF BIOLOGICAL CHEMISTRY 267: 546 (1992).

4. Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein Precursor

5. CHARTIERHARLIN, M.C., EARLY-ONSET ALZHEIMERS-DISEASE CAUSED BY MUTATIONS AT CODON-717 OF THE BETA-AMYLOID PRECURSOR PROTEIN GENE, NATURE 353: 844 (1991).

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