Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein Precursor
Author:
Affiliation:
1. Division of Neuropathology, Institute of Pathology, Case Western Reserve University, Cleveland, OH 44106.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference36 articles.
1. BURDICK, D, ASSEMBLY AND AGGREGATION PROPERTIES OF SYNTHETIC ALZHEIMERS A4/BETA AMYLOID PEPTIDE ANALOGS, JOURNAL OF BIOLOGICAL CHEMISTRY 267: 546 (1992).
2. CAI X.D. unpublished data.
3. CAPORASO, G.L., CHLOROQUINE INHIBITS INTRACELLULAR DEGRADATION BUT NOT SECRETION OF ALZHEIMER BETA/A4 AMYLOID PRECURSOR PROTEIN, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 89: 2252 (1992).
4. CHARTIERHARLIN, M.C., EARLY-ONSET ALZHEIMERS-DISEASE CAUSED BY MUTATIONS AT CODON-717 OF THE BETA-AMYLOID PRECURSOR PROTEIN GENE, NATURE 353: 844 (1991).
5. Cole, G. M., Molecular Biology and Genetics of Alzheimer's Disease: 113 (1990).
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