Slc4a11 Gene Disruption in Mice-Reference-Cited by-同舟云学术

Slc4a11 Gene Disruption in Mice

Published:2009-09 Issue:39 Volume:284 Page:26882-26896
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Lopez Ivan A.,Rosenblatt Mark I.,Kim Charles,Galbraith Gary C.,Jones Sherri M.,Kao Liyo,Newman Debra,Liu Weixin,Yeh Stacey,Pushkin Alexander,Abuladze Natalia,Kurtz Ira

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference44 articles.

1. SLC4 base (HCO3−, CO32−) transporters: classification, function, structure, genetic diseases, and knockout models

2. Human BTR1, a New Bicarbonate Transporter Superfamily Member and Human AE4 from Kidney

3. NaBC1 Is a Ubiquitous Electrogenic Na+-Coupled Borate Transporter Essential for Cellular Boron Homeostasis and Cell Growth and Proliferation

4. Arabidopsis boron transporter for xylem loading

5. Mutations in sodium-borate cotransporter SLC4A11 cause recessive congenital hereditary endothelial dystrophy (CHED2)

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1. NH3/NH4+ allosterically activates SLC4A11 by causing an acidic shift in the intracellular pK that governs H+(OH−) conductance;Frontiers in Physiology;2024-08-14

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3. Animal Models in Eye Research: Focus on Corneal Pathologies;International Journal of Molecular Sciences;2023-11-23

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