Post-translational Disruption of the ΔF508 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)-Molecular Chaperone Complex with Geldanamycin Stabilizes ΔF508 CFTR in the Rabbit Reticulocyte Lysate-Reference-Cited by-同舟云学术

Post-translational Disruption of the ΔF508 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR)-Molecular Chaperone Complex with Geldanamycin Stabilizes ΔF508 CFTR in the Rabbit Reticulocyte Lysate

Published:2000-12 Issue:48 Volume:275 Page:37462-37468
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Fuller William,Cuthbert Alan W.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

1. Identification of the Cystic Fibrosis Gene: Cloning and Characterization of Complementary DNA

2. Identification of the Cystic Fibrosis Gene: Genetic Analysis

3. Expression of cystic fibrosis transmembrane conductance regulator corrects defective chloride channel regulation in cystic fibrosis airway epithelial cells

4. Generation of cAMP-Activated Chloride Currents by Expression of CFTR

5. Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis

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