Mutant Presenilin 1 Increases the Expression and Activity of BACE1-Reference-Cited by-同舟云学术

Mutant Presenilin 1 Increases the Expression and Activity of BACE1

Published:2009-04 Issue:14 Volume:284 Page:9027-9038
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Giliberto Luca,Borghi Roberta,Piccini Alessandra,Mangerini Rosa,Sorbi Sandro,Cirmena Gabriella,Garuti Anna,Ghetti Bernardino,Tagliavini Fabrizio,Mughal Mohamed R.,Mattson Mark P.,Zhu Xiongwei,Wang Xinglong,Guglielmotto Michela,Tamagno Elena,Tabaton Massimo

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference65 articles.

1. Amyloid β-peptide is produced by cultured cells during normal metabolism

2. Two transmembrane aspartates in presenilin-1 required for presenilin endoproteolysis and γ-secretase activity

3. Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

4. Enhanced Production and Oligomerization of the 42-residue Amyloid β-Protein by Chinese Hamster Ovary Cells Stably Expressing Mutant Presenilins

5. Presence of soluble amyloid β–peptide precedes amyloid plaque formation in Down's syndrome

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