IL-11 ameliorates oxidative stress damage in neurons after spinal cord injury by activating the JAK/STAT signaling pathway-Reference-Cited by-同舟云学术

IL-11 ameliorates oxidative stress damage in neurons after spinal cord injury by activating the JAK/STAT signaling pathway

Published:2024-01 Issue: Volume:127 Page:111367
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Sun Yang,Song Xue,Geng Zhijun,Xu Yibo,Xiao Linyu,Chen Yue,Li Bohan,Shi Jinran,Wang Lian,Wang Yueyue,Zhang Xiaofeng,Zuo Lugen,Li Jing,Lü Hezuo,Hu Jianguo^ORCID

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference35 articles.

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2. The future of artificial hibernation medicine: Protection of nerves and organs after spinal cord injury;Liu;Neural. Regen. Res.,2024

3. Research progress on the application of transcranial magnetic stimulation in spinal cord injury rehabilitation: A narrative review;Wang;Front. Neurol.,2023

4. Efficacy of rehabilitation interventions evaluated in common neurological conditions in improving participation outcomes: A systematic review;Kossi;Clin. Rehabil.,2023

5. Oxidative stress following spinal cord injury: From molecular mechanisms to therapeutic targets;Yu;J. Neurosci. Res.,2023

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1. Biomineralized MnO2 Nanoparticle-Constituted Hydrogels Promote Spinal Cord Injury Repair by Modulating Redox Microenvironment and Inhibiting Ferroptosis;Pharmaceutics;2024-08-12

2. NF-κB and JAK/STAT Signaling Pathways as Crucial Regulators of Neuroinflammation and Astrocyte Modulation in Spinal Cord Injury;Cells;2024-03-26