Inhibition of Interleukin-17A, But Not Interleukin-17F, Signaling Lowers Blood Pressure, and Reduces End-Organ Inflammation in Angiotensin II–Induced Hypertension-Reference-Cited by-同舟云学术

Inhibition of Interleukin-17A, But Not Interleukin-17F, Signaling Lowers Blood Pressure, and Reduces End-Organ Inflammation in Angiotensin II–Induced Hypertension

Published:2016-12 Issue:7 Volume:1 Page:606-616
ISSN:2452-302X
Container-title:JACC: Basic to Translational Science
language:en
Short-container-title:JACC: Basic to Translational Science

Author:

Saleh Mohamed A.,Norlander Allison E.,Madhur Meena S.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference32 articles.

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2. Inflammation, immunity, and hypertensive end-organ damage;McMaster;Circ Res,2015

3. Interleukin 17 promotes angiotensin II-induced hypertension and vascular dysfunction;Madhur;Hypertension,2010

4. Interleukin-17 causes Rho-kinase-mediated endothelial dysfunction and hypertension;Nguyen;Cardiovasc Res,2013

5. Renal transporter activation during angiotensin-II hypertension is blunted in interferon-gamma-/- and interleukin-17A-/- mice;Kamat;Hypertension,2015

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