Genetic variants at the 9p21 locus contribute to atherosclerosis through modulation of ANRIL and CDKN2A/B-Reference-Cited by-同舟云学术

Genetic variants at the 9p21 locus contribute to atherosclerosis through modulation of ANRIL and CDKN2A/B

Published:2012-02 Issue:2 Volume:220 Page:449-455
ISSN:0021-9150
Container-title:Atherosclerosis
language:en
Short-container-title:Atherosclerosis

Author:

Congrains Ada,Kamide Kei,Oguro Ryousuke,Yasuda Osamu,Miyata Keishi,Yamamoto Eiichiro,Kawai Tatsuo,Kusunoki Hiroshi,Yamamoto Hiroko,Takeya Yasushi,Yamamoto Koichi,Onishi Miyuki,Sugimoto Ken,Katsuya Tomohiro,Awata Nobuhisa,Ikebe Kazunori,Gondo Yasuyuki,Oike Yuichi,Ohishi Mitsuru,Rakugi Hiromi

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

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2. Susceptibility to coronary artery disease and diabetes is encoded by distinct, tightly linked, SNPs in the ANRIL locus on chromosome 9p;Broadbent;Hum Mol Genet,2008

3. Regulation of the INK4b–ARF–INK4a tumour suppressor locus: all for one or one for all;Gil1;Nat Rev Mol Cell Biol,2006

4. Molecular Interplay of the noncoding RNA ANRIL and methylated histone H3 lysine 27 by Polycomb CBX7 in transcriptional silencing of INK4a;Yap;Mol Cell,2010

5. Long non-coding RNA ANRIL is required for the PRC2 recruitment to and silencing of p15INK4B tumor suppressor gene;Kotake;Oncogene,2011

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