T cell-specific BLIMP-1 deficiency exacerbates experimental autoimmune encephalomyelitis in nonobese diabetic mice by increasing Th1 and Th17 cells-Reference-Cited by-同舟云学术

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T cell-specific BLIMP-1 deficiency exacerbates experimental autoimmune encephalomyelitis in nonobese diabetic mice by increasing Th1 and Th17 cells

Published:2014-04 Issue:2 Volume:151 Page:101-113
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Lin Ming-Hong^ORCID,Yeh Li-Tzu^ORCID,Chen Shyi-Jou,Chiou Hsin-Ying C.,Chu Chin-Chen,Yen Linju B.,Lin Kuo-I,Chang Deh-Ming^ORCID,Sytwu Huey-Kang

Funder

National Science Council, Taiwan, ROC

Tri-service General Hospital

Chi Mei Medical Center

C.Y. Foundation for Advancement of Education, Sciences and Medicine

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference50 articles.

1. Blimp-1, a novel zinc finger-containing protein that can drive the maturation of B lymphocytes into immunoglobulin-secreting cells;Turner;Cell,1994

2. Repression of c-myc transcription by Blimp-1, an inducer of terminal B cell differentiation;Lin;Science,1997

3. BCL-6 represses genes that function in lymphocyte differentiation, inflammation, and cell cycle control;Shaffer;Immunity,2000

4. Blimp-1-dependent repression of Pax-5 is required for differentiation of B cells to immunoglobulin M-secreting plasma cells;Lin;Mol. Cell. Biol.,2002

5. Transcriptional repressor Blimp-1 is essential for T cell homeostasis and self-tolerance;Kallies;Nat. Immunol.,2006

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