Sclerostin antibody (Scl-Ab) improves osteomalacia phenotype in dentin matrix protein 1(Dmp1) knockout mice with little impact on serum levels of phosphorus and FGF23-Reference-Cited by-同舟云学术

Sclerostin antibody (Scl-Ab) improves osteomalacia phenotype in dentin matrix protein 1(Dmp1) knockout mice with little impact on serum levels of phosphorus and FGF23

Published:2016-05 Issue: Volume:52-54 Page:151-161
ISSN:0945-053X
Container-title:Matrix Biology
language:en
Short-container-title:Matrix Biology

Author:

Ren Yinshi^ORCID,Han Xianglong,Jing Yan,Yuan Baozhi,Ke Huazhu,Liu Min,Feng Jian Q.

Funder

U.S. National Institutes of Health

National Natural Science Foundation of China

Amgen and UCB Pharma Research

Publisher

Elsevier BV

Subject

Molecular Biology

Reference45 articles.

1. Characterization of a novel dentin matrix acidic phosphoprotein. Implications for induction of biomineralization;George;J. Biol. Chem.,1993

2. Dentin matrix protein 1 is predominantly expressed in chicken and rat osteocytes but not in osteoblasts;Toyosawa;J. Bone Miner. Res.,2001

3. Dentin matrix protein 1, a target molecule for Cbfa1 in bone, is a unique bone marker gene;Fen;J. Bone Miner. Res.,2002

4. Dmp1-deficient mice display severe defects in cartilage formation responsible for a chondrodysplasia-like phenotype;Ye;J. Biol. Chem.,2005

5. Deletion of dentin matrix protein-1 leads to a partial failure of maturation of predentin into dentin, hypomineralization, and expanded cavities of pulp and root canal during postnatal tooth development;Ye;J. Biol. Chem.,2004

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