A developmental redox dysregulation leads to spatio-temporal deficit of parvalbumin neuron circuitry in a schizophrenia mouse model

Author:

Cabungcal Jan-Harry,Steullet Pascal,Kraftsik Rudolf,Cuenod Michel,Do Kim Q.

Funder

Swiss National Science Foundation

Damm-Etienne Foundation

Alamaya Foundation

Publisher

Elsevier BV

Subject

Biological Psychiatry,Psychiatry and Mental health

Reference80 articles.

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3. Multiple risk pathways for schizophrenia converge in serine racemase knockout mice, a mouse model of NMDA receptor hypofunction;Balu;Proc. Natl. Acad. Sci. U. S. A.,2013

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5. Synaptic NMDA receptor activity is coupled to the transcriptional control of the glutathione system;Baxter;Nat. Commun.,2015

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