E-Cadherin is Dispensable to Maintain Langerhans Cells in the Epidermis-Reference-Cited by-同舟云学术

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E-Cadherin is Dispensable to Maintain Langerhans Cells in the Epidermis

Published:2020-01 Issue:1 Volume:140 Page:132-142.e3
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Brand Anna,Diener Nathalie,Zahner Sonja P.,Tripp Christoph,Backer Ronald A.,Karram Khalad,Jiang Aimin,Mellman Ira,Stoitzner Patrizia,Clausen Björn E.

Funder

Landsteiner Foundation for Blood Transfusion Research

LSBR

Netherlands Organisation for Scientific Research

NWO

German Science Foundation

DFG

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference61 articles.

1. Immunoregulatory functions of KLRG1 cadherin interactions are dependent on forward and reverse signaling;Banh;Blood,2009

2. Characterization of mouse CD4 T cell subsets defined by expression of KLRG1;Beyersdorf;Eur J Immunol,2007

3. Autocrine/paracrine TGF-beta1 inhibits Langerhans cell migration;Bobr;Proc Natl Acad Sci U S A,2012

4. Expression of E-cadherin by murine dendritic cells: E-cadherin as a dendritic cell differentiation antigen characteristic of epidermal Langerhans cells and related cells;Borkowski;Eur J Immunol,1994

5. E-cadherin is a survival factor for the lactating mouse mammary gland;Boussadia;Mech Dev,2002

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3. Influence of E‐cadherin deficiency on the dendrite morphology of Langerhans cells;European Journal of Immunology;2024-05-22

4. Review: A Contemporary, Multifaced Insight into Psoriasis Pathogenesis;Journal of Personalized Medicine;2024-05-16

5. Serine protease Rv2569c facilitates transmission of Mycobacterium tuberculosis via disrupting the epithelial barrier by cleaving E-cadherin;PLOS Pathogens;2024-05-09

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