Intracellular protein degradation in cultures of dystrophic muscle cells and fibroblasts
Author:
Publisher
Elsevier BV
Subject
Cell Biology
Reference41 articles.
1. Increased myofibrillar protein catabolism in Duchenne muscular dystrophy measured by 3-methylhistidine excretion in the urine.
2. Increased Turnover of Muscle Contractile Proteins in Duchenne Muscular Dystrophy as Assessed by 3-Methylhistidine and Creatinine Excretion
3. Comparison of the turnover patterns of total and individual muscle proteins in normal mice and those with hereditary muscular dystrophy
4. Pathogenesis of human muscular dystrophies;Goldberg,1977
5. Skeletal muscle protein and amino acid metabolism in hereditary mouse muscular dystrophy. The role of disordered cyclic nucleotide metabolism in the accelerated alanine and glutamine formation and release.
Cited by 9 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Effects of a beta-adrenergic agonist on protein turnover in muscle cells in culture;Biochemical Pharmacology;1989-07
2. Regulation of protein synthesis and degradation in L8 myotubes. Effects of serum, insulin and insulin-like growth factors;Biochemical Journal;1989-06-01
3. Histidine and Proline are Important Sites of Free Radical Damage to Proteins;Free Radical Research Communications;1989-01
4. Cell growth and substrate effects on characteristics of a lysosomal enzyme (cathepsin C) in Duchenne muscular dystrophy fibroblasts;Biochemistry and Cell Biology;1987-07-01
5. Abnormal protein metabolism in skin fibroblasts invitro from patients with Duchenne muscular dystrophy;Biochemical and Biophysical Research Communications;1987-05
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