Abnormal protein metabolism in skin fibroblasts invitro from patients with Duchenne muscular dystrophy
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference37 articles.
1. The metabolic basis of inherited disease;Appel,1978
2. Increased Turnover of Muscle Contractile Proteins in Duchenne Muscular Dystrophy as Assessed by 3-Methylhistidine and Creatinine Excretion
3. Increased myofibrillar protein catabolism in Duchenne muscular dystrophy measured by 3-methylhistidine excretion in the urine.
4. Increased rates of myofibrillar protein breakdown in muscle-wasting diseases
5. Biochemistry of muscle membranes in Duchenne muscular dystrophy
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1. Power line frequency electromagnetic fields do not increase the rate of protein synthesis in human skin fibroblasts as previously reported;Bioelectromagnetics;2003-08-27
2. Differential Gene Expression, Protein Synthesis, and Degradation in Aging Fibroblasts;Cell and Tissue Culture Models in Dermatological Research;1993
3. Fibroblasts in normal and pathological terminal differentiation, aging, apoptosis and transformation;Archives of Gerontology and Geriatrics;1992-01
4. Degradation of individual intracellular proteins analyzed by two-dimensional gel electrophoresis and computerized video densitometry;Electrophoresis;1990
5. Differential degradation of intracellular proteins in human skin fibroblasts of mitotic and mitomycin-C (MMC)-induced postmitotic differentiation states in vitro;Differentiation;1989-10
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