Rab7 reduces α-synuclein toxicity in rats and primary neurons-Reference-Cited by-同舟云学术

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Rab7 reduces α-synuclein toxicity in rats and primary neurons

Published:2022-01 Issue: Volume:347 Page:113900
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Szegö Eva M.,Van den Haute Chris,Höfs Lennart,Baekelandt Veerle,Van der Perren Anke,Falkenburger Björn H.

Funder

Queen Elisabeth Medical Foundation

DFG

European Coordinated Research on Long-term Challenges in Information and Communication Sciences & Technologies ERA-NET

FWO

KU Leuven

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference73 articles.

1. RAB7 controls melanoma progression by exploiting a lineage-specific wiring of the endolysosomal pathway;Alonso-Curbelo;Cancer Cell,2014

2. Phosphorylation of Ser-129 is the dominant pathological modification of α-synuclein in familial and sporadic lewy body disease;Anderson;J. Biol. Chem.,2006

3. Phosphatidylinositol 4,5-bisphosphate controls Rab7 and PLEKHM 1 membrane cycling during autophagosome–lysosome fusion;Baba;EMBO J.,2019

4. Characterization of lentiviral vector-mediated gene transfer in adult mouse brain;Baekelandt;Hum. Gene Ther.,2002

5. The endocytic membrane trafficking pathway plays a major role in the risk of Parkinson’s disease;Bandres-Ciga;Mov. Disord.,2019

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1. Evaluation of the Effect of β-Wrapin AS69 in a Mouse Model Based on Alpha-Synuclein Overexpression;Biomolecules;2024-06-25

2. Application of OpenArray Technology to Assess Changes in the Expression of Functionally Significant Genes in the Substantia Nigra of Mice in a Model of Parkinson’s Disease;Genes;2023-12-12

3. Multiple tethers of organelle contact sites are involved in α-synuclein toxicity in yeast;Molecular Biology of the Cell;2023-07-01

4. Potent New Targets for Autophagy Enhancement to Delay Neuronal Ageing;Cells;2023-06-30

5. Disrupting the α-synuclein-ESCRT interaction with a peptide inhibitor mitigates neurodegeneration in preclinical models of Parkinson’s disease;Nature Communications;2023-04-19

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