Induction of sestrin2 as an endogenous protective mechanism against amyloid beta-peptide neurotoxicity in primary cortical culture-Reference-Cited by-同舟云学术

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Induction of sestrin2 as an endogenous protective mechanism against amyloid beta-peptide neurotoxicity in primary cortical culture

Published:2014-03 Issue: Volume:253 Page:63-71
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Chen Yueh-Sheng,Chen Shang-Der,Wu Chia-Lin,Huang Shiang-Suo,Yang Ding-I

Funder

Chang Gung Memorial Hospital

Ministry of Education in Taiwan-Aim for the Top University Plan

National Science Council

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference43 articles.

1. Sestrins activate Nrf2 by promoting p62-dependent autophagic degradation of Keap1 and prevent oxidative liver damage;Bae;Cell Metab.,2013

2. Hydrogen peroxide mediates amyloid beta protein toxicity;Behl;Cell,1994

3. Nelfinavir and bortezomib inhibit mTOR activity via ATF4-mediated sestrin-2 regulation;Bruning;Mol. Oncol.,2013

4. Stress-responsive sestrins link p53 with redox regulation and mammalian target of rapamycin signaling;Budanov;Antioxid. Redox Signal.,2011

5. Identification of a novel stress-responsive gene Hi95 involved in regulation of cell viability;Budanov;Oncogene,2002

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