Sestrin2 can alleviate endoplasmic reticulum stress to improve traumatic brain injury by activating AMPK/mTORC1 signaling pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Biochemistry
Link
https://link.springer.com/content/pdf/10.1007/s11011-023-01323-2.pdf
Reference90 articles.
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4. Cangelosi AL, Puszynska AM, Roberts JM, Armani A, Nguyen TP, Spinelli JB, Kunchok T, Wang B, Chan SH, Lewis CA, Comb WC, Bell GW, Helman A, Sabatini DM (2022) Zonated leucine sensing by Sestrin-mTORC1 in the liver controls the response to dietary leucine. Science 377(6601):47–56. https://doi.org/10.1126/science.abi9547
5. Chantranupong L, Wolfson RL, Orozco JM, Saxton RA, Scaria SM, Bar-Peled L, Spooner E, Isasa M, Gygi SP, Sabatini DM (2014) The sestrins interact with GATOR2 to negatively regulate the amino-acid-sensing pathway upstream of mTORC1. Cell Rep 9(1):1–8. https://doi.org/10.1016/j.celrep.2014.09.014
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2. Endoplasmic reticulum stress and the unfolded protein response: emerging regulators in progression of traumatic brain injury;Cell Death & Disease;2024-02-20
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