IL36RN mutations define a severe autoinflammatory phenotype of generalized pustular psoriasis-Reference-Cited by-同舟云学术

IL36RN mutations define a severe autoinflammatory phenotype of generalized pustular psoriasis

Published:2015-04 Issue:4 Volume:135 Page:1067-1070.e9
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Hussain Safia,Berki Dorottya M.,Choon Siew-Eng,Burden A. David,Allen Michael H.,Arostegui Juan I.,Chaves Antonio,Duckworth Michael,Irvine Alan D.,Mockenhaupt Maja,Navarini Alexander A.,Seyger Marieke M.B.,Soler-Palacin Pere,Prins Christa,Valeyrie-Allanore Laurence,Vicente M. Asuncion,Trembath Richard C.,Smith Catherine H.,Barker Jonathan N.,Capon Francesca

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference34 articles.

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2. Psoriasis;Griffiths,2010

3. Interleukin-36-receptor antagonist deficiency and generalized pustular psoriasis;Marrakchi;N Engl J Med,2011

4. Mutations in IL36RN/IL1F5 are associated with the severe episodic inflammatory skin disease known as generalized pustular psoriasis;Onoufriadis;Am J Hum Genet,2011

5. Inflammation: IL-36 has proinflammatory effects in skin but not in joints;Dietrich;Nat Rev Rheumatol,2014

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