IL-36 has proinflammatory effects in skin but not in joints
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://www.nature.com/articles/nrrheum.2014.156.pdf
Reference10 articles.
1. Derer, A. et al. Blockade of IL-36 receptor signaling does not prevent from TNF-induced arthritis. PLoS ONE 9, e101954 (2014).
2. Sims J. E., Vigne S., Gabay C. & Towne J. in Cytokine Frontiers: Regulation of Immune Response in Health and Disease. (eds Yoshimoto, T. & Yoshimoto, T.) 199–214 (Springer, 2014).
3. Vigne, S. et al. IL-36R ligands are potent regulators of dendritic and T cells. Blood 118, 5813–5823 (2011).
4. Foster, A. M. et al. IL-36 promotes myeloid cell infiltration, activation, and inflammatory activity in skin. J. Immunol. 192, 6053–6061 (2014).
5. Vigne, S. et al. IL-36 signaling amplifies TH1 responses by enhancing proliferation and TH1 polarization of naive CD4+ T cells. Blood 120, 3478–3487 (2012).
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