Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons-Reference-Cited by-同舟云学术

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Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons

Published:2022-08 Issue: Volume:12 Page:100109
ISSN:2452-073X
Container-title:Neurobiology of Pain
language:en
Short-container-title:Neurobiology of Pain

Author:

Loya-López Santiago I.,Duran Paz,Ran Dongzhi,Calderon-Rivera Aida,Gomez Kimberly,Moutal Aubin,Khanna Rajesh

Funder

National Institutes of Health

National Institute of Neurological Disorders and Stroke

Publisher

Elsevier BV

Subject

Anesthesiology and Pain Medicine,Neurology (clinical),Neuroscience (miscellaneous)

Reference57 articles.

1. Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons;Ahn;Mol. Pain,2011

2. Prostaglandin E2 sensitizes the cough reflex centrally via EP3 receptor-dependent activation of NaV 1.8 channels;Al-Kandery;Respir. Res.,2021

3. Phosphorylation of collapsin response mediator protein-2 by Rho-kinase. Evidence for two separate signaling pathways for growth cone collapse;Arimura;J. Biol. Chem.,2000

4. Phosphorylation by Rho kinase regulates CRMP-2 activity in growth cones;Arimura;Mol. Cell. Biol.,2005

5. The role of voltage-gated sodium channels in pain signaling;Bennett;Physiol. Rev.,2019

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. 20S-Ginsenoside Rh2, the major bioactive saponin in Panax notoginseng flowers, ameliorates cough by inhibition of NaV1.7 and TRPV1 channel currents and downregulation of TRPV1 expression;Journal of Ethnopharmacology;2025-01

2. Small molecule targeting NaV1.7 via inhibition of CRMP2-Ubc9 interaction reduces pain-related outcomes in a rodent osteoarthritic model;Pain;2024-07-26

3. Intranasal CRMP2-Ubc9 inhibitor regulates NaV1.7 to alleviate trigeminal neuropathic pain;Pain;2023-09-26

4. Intranasal CRMP2-Ubc9 Inhibitor Regulates Na_V1.7 to Alleviate Trigeminal Neuropathic Pain;2023-07-17

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