Kindlin-2 interacts with and stabilizes DNMT1 to promote breast cancer development-Reference-Cited by-同舟云学术

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Kindlin-2 interacts with and stabilizes DNMT1 to promote breast cancer development

Published:2018-12 Issue: Volume:105 Page:41-51
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Wang Peng,Chu Wenhui,Zhang Xi,Li Bing,Wu Junzhou,Qi Lihua,Yu Yu,Zhang Hongquan^ORCID

Funder

Ministry of Science and Technology of the People's Republic of China

National Natural Science Foundation of China

Natural Science Foundation of Beijing Municipality

111 Project of the Ministry of Education of China, Peking University

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference43 articles.

1. Increased protein stability causes DNA methyltransferase 1 dysregulation in breast cancer;Agoston;J. Biol. Chem.,2005

2. Alterations in DNA methylation: a fundamental aspect of neoplasia;Baylin;Adv. Cancer Res.,1998

3. The transcription factor Slug represses E-cadherin expression and induces epithelial to mesenchymal transitions: a comparison with Snail and E47 repressors;Bolos;J. Cell. Sci.,2003

4. Methylation of the E-cadherin gene in bladder neoplasia and in normal urothelial epithelium from elderly individuals;Bornman;Am. J. Pathol.,2001

5. Mechanisms associated with abnormal E-cadherin immunoreactivity in human bladder tumors;Bringuier;Int. J. Cancer J. Int. du Cancer,1999

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