Mitochondrial toxins in models of neurodegenerative diseases. II: elevated zif268 transcription and independent temporal regulation of striatal D1 and D2 receptor mRNAs and D1 and D2 receptor-binding sites in C57BL/6 mice during MPTP treatment-Reference-Cited by-同舟云学术

Mitochondrial toxins in models of neurodegenerative diseases. II: elevated zif268 transcription and independent temporal regulation of striatal D1 and D2 receptor mRNAs and D1 and D2 receptor-binding sites in C57BL/6 mice during MPTP treatment

Published:1997-08 Issue:2 Volume:765 Page:189-197
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Smith Trisha S,Trimmer Patricia A,Khan Shaharyar M,Tinklepaugh David L,Bennett James P

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference41 articles.

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2. Selective nigral toxicity after systemic administration of 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine (MPTP) in the squirrel monkey;Langston;Brain Res.,1984

3. 1-Methyl-4-phenylpyridinium ion (MPP+): identification of a metabolite of MPTP, a toxin selective to the substantia nigra;Langston;Neurosci. Lett.,1984

4. Parkinsonism-inducing neurotoxin 1-methyl-4-phenyl-1,2,3,6 tetrahydropyridine: uptake of the metabolite 1-methyl-4-phenyl-pyridinium by dopamine neurons explains selective toxicity;Javich;Proc. Natl. Acad. Sci. USA,1985

5. Selective retention of MPP+ within the monoaminergic systems of the primate brain following MPTP administration: an in vivo autoradiographic study;Herkenham;Neuroscience,1991

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