“New” hepatic fat activates PPARα to maintain glucose, lipid, and cholesterol homeostasis-Reference-Cited by-同舟云学术

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“New” hepatic fat activates PPARα to maintain glucose, lipid, and cholesterol homeostasis

Published:2005-05 Issue:5 Volume:1 Page:309-322
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Chakravarthy Manu V.,Pan Zhijun,Zhu Yimin,Tordjman Karen,Schneider Jochen G.,Coleman Trey,Turk John,Semenkovich Clay F.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference60 articles.

1. The mechanisms of action of PPARs;Berger;Annu. Rev. Med.,2002

2. Respiratory uncoupling lowers blood pressure through a leptin-dependent mechanism in genetically obese mice;Bernal-Mizrachi;Arterioscler. Thromb. Vasc. Biol.,2002

3. Dexamethasone induction of hypertension and diabetes is PPAR-alpha dependent in LDL receptor-null mice;Bernal-Mizrachi;Nat. Med.,2003

4. Increased pyruvate flux capacities account for diet-induced increases in gluconeogenesis in vitro;Bizeau;Am. J. Physiol. Regul. Integr. Comp. Physiol.,2001

5. Central role for liver X receptor in insulin-mediated activation of SREBP-1c transcription and stimulation of fatty acid synthesis in liver;Chen;Proc. Natl. Acad. Sci. USA,2004

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