The effect of the voltage-gated sodium channel NaV1.7 blocker PF-05089771 on cough in the guinea pig
Author:
Funder
Scientific Grant Agency of the MŠVVaŠ and the SAV
Publisher
Elsevier BV
Subject
Pulmonary and Respiratory Medicine,Physiology,General Neuroscience
Reference14 articles.
1. Subtype-selective small molecule inhibitors reveal a fundamental role for Nav1.7 in Nociceptor Electrogenesis, axonal conduction and presynaptic release;Alexandrou;PLoS One,2016
2. The role of voltage-gated sodium channels in pain signaling;Bennett;Physiol. Rev.,2019
3. Comparison of TRPA1-versus TRPV1-mediated cough in guinea pigs;Brozmanova;Eur. J. Pharmacol.,2012
4. The voltage-gated sodium channel NaV1.8 blocker A-803467 inhibits cough in the guinea pig;Brozmanova;Respir. Physiol. Neurobiol.,2019
5. Antitussive drugs—past, present, and future;Dicpinigaitis;Pharmacol. Rev.,2014
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1. 20S-Ginsenoside Rh2, the major bioactive saponin in Panax notoginseng flowers, ameliorates cough by inhibition of NaV1.7 and TRPV1 channel currents and downregulation of TRPV1 expression;Journal of Ethnopharmacology;2025-01
2. Influence of combined voltage-gated sodium channel NaV1.7 and NaV1.8 inhibitors on cough in a guinea pig model;Respiratory Physiology & Neurobiology;2023-06
3. NaV1.7 channels are expressed in the lower airways of the human respiratory tract;Respiratory Physiology & Neurobiology;2023-05
4. Mechanism of electroacupuncture at acupoints of the lung meridian through PKA/PKC regulation of TRPV1 in chronic cough after lung surgery in guinea pigs;Journal of Thoracic Disease;2023-04
5. Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons;Neurobiology of Pain;2022-08
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