Extracellular Forms of Aβ and Tau from iPSC Models of Alzheimer’s Disease Disrupt Synaptic Plasticity-Reference-Cited by-同舟云学术

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Extracellular Forms of Aβ and Tau from iPSC Models of Alzheimer’s Disease Disrupt Synaptic Plasticity

Published:2018-05 Issue:7 Volume:23 Page:1932-1938
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Hu Neng-Wei,Corbett Grant T.,Moore Steven,Klyubin Igor,O’Malley Tiernan T.,Walsh Dominic M.,Livesey Frederick J.,Rowan Michael J.

Funder

Science Foundation Ireland

Wellcome Trust

Alborada Trust

Alzheimer’s Research UK Stem Cell Research Centre

NIH

Cancer Research UK

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference31 articles.

1. The toxic Aβ oligomer and Alzheimer’s disease: an emperor in need of clothes;Benilova;Nat. Neurosci.,2012

2. Human secreted tau increases amyloid-beta production;Bright;Neurobiol. Aging,2015

3. Extracellular tau oligomers produce an immediate impairment of LTP and memory;Fá;Sci. Rep.,2016

4. The ordered assembly of tau is the gain-of-toxic function that causes human tauopathies;Goedert;Alzheimers Dement.,2016

5. Probing the secrets of Alzheimer’s disease using human-induced pluripotent stem cell technology;Goldstein;Neurotherapeutics,2015

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