DNA-PK inhibition causes a low level of H2AX phosphorylation and homologous recombination repair in Medaka (Oryzias latipes) cells
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference27 articles.
1. H2AX is required for cell cycle arrest via the p53/p21 pathway;Fragkos;Mol. Cell Biol.,2009
2. GammaH2AX and cancer;Bonner;Nat. Rev. Cancer,2008
3. Current topics in DNA double-strand break repair;Kobayashi;J. Radiat. Res. (Tokyo),2008
4. The life and death of DNA-PK;Collis;Oncogene,2005
5. Ku DNA end-binding protein modulates homologous repair of double-strand breaks in mammalian cells;Pierce;Genes Dev.,2001
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2. Evaluation of interactive effects of phosphorus-32 and copper on marine and freshwater bivalve mollusks;International Journal of Radiation Biology;2020-10-06
3. CC-115, a dual inhibitor of mTOR kinase and DNA-PK, blocks DNA damage repair pathways and selectively inhibits ATM-deficient cell growth in vitro;Oncotarget;2017-08-18
4. Reprogramming progeria fibroblasts re-establishes a normal epigenetic landscape;Aging Cell;2017-06-08
5. An Approach to Elucidate NBS1 Function in DNA Repair Using Frequent Nonsynonymous Polymorphism in Wild Medaka (Oryzias latipes) Populations;PLOS ONE;2017-01-20
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